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A hyperprostaglandin E syndrome mutation in Kir1.1 (renal outer medullary potassium) channels reveals a crucial residue for channel function in Kir1.3 channels.
Authors: Derst, C.; Wischmeyer, E.; Preisig-Mueller, R.; Spauschus, A.; Konrad, M.; Hensen, P.; Jeck, N.; Seyberth, H. W.; Daut, J.; Karschin, A.
Date of Publication (YYYY-MM-DD): 1998
Title of Journal: Journal of Biological Chemistry
Volume: 273
Start Page: 23884
End Page: 23891
Document Type: Article
ID: 233195.0
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