Fritsch, A., J. S. Kern, S. Loeckermann, D. Velati, R. Fässler and L. Bruckner-Tuderman: Conditional collagen VII inactivation allows analysis of anchoring fibril stability and function in vivo and reveals a major role of fibroblasts in collagen VII expression. In: Journal of Investigative Dermatology 129, Suppl. 1, S81-S81, Seq. No.: 483 (2009).
Fritsch, A., J. S. Kern, S. Loeckermann, D. Velati, R. Fässler and L. Bruckner-Tuderman: Conditional collagen VII inactivation reveals high anchoring fibril stability in vivo: implications for molecular therapies. In: Experimental Dermatology 18, 3, 282-282 (2009).
Has, C., R. J. Ludwig, C. Herz, J. S. Kern, S. Ussar, F. R. Ochsendorf, R. Kaufmann, H. Schumann, J. Kohlhase and L. Bruckner-Tuderman: C-terminally truncated kindlin-1 leads to abnormal adhesion and migration of keratinocytes. In: British Journal of Dermatology 159, 5, 1192-1196 (2008).
Fritsch, A., S. Loeckermann, J. S. Kern, A. Braun, M. R. Bösl, T. A. Bley, H. Schumann, D. von Elverfeldt, D. Paul, M. Erlacher, D. B. von Rautenfeld, I. Hausser, R. Fässler and L. Bruckner-Tuderman: A hypomorphic mouse model of dystrophic epidermolysis bullosa reveals mechanisms of disease and response to fibroblast therapy. In: Journal of Clinical Investigation 118, 5, 1669-1679 (2008).
Fritsch, A., S. Loeckermann, J. S. Kern, H. Schumann, R. Fässler and L. Bruckner-Tuderman: Molecular analysis of mitten deformities in the collagen VII hypomorph and human RDEB reveals potential targets for therapy. In: JOURNAL OF INVESTIGATIVE DERMATOLOGY 128, Suppl. Suppl. 1, S122-S122, Seq. No.: 731 (2008).
Kern, J. S., S. Loeckermann, A. Fritsch, M. L. Muller, R. Fässler and L. Bruckner-Tuderman: Cell therapy restores skin function in an immunocompetent mouse model for dystrophic epidermolysis bullosa. In: JOURNAL OF INVESTIGATIVE DERMATOLOGY 128, Suppl. Suppl. 1, S116-S116, Seq. No.: 692 (2008).
Kern, J. S., S. Lockermann, A. Fritsch, R. Fässler and L. Bruckner-Tuderman: Cell therapy restores skin function in an immunocompetent mouse model for dystrophic epidermolysis bullosa. In: Experimental Dermatology 17, 3, 249-249 (2008).
Fritsch, A., S. Lockermann, J. S. Kern, R. Faessler and L. Bruckner-Tuderman: The collagen VII hypomorph develops pseudosyndactyly, a key symptom of recessive epidermolyis bullosa, as a result of aberrant contractile fibrosis-implications for therapy. In: Experimental Dermatology 17, 3, 245-245 (2008).
Fritsch, A., S. Loeckermann, J. S. Kern, R. Fässler and L. Bruckner-Tuderman: The collagen VII hypomorph, a viable mouse model for dystrophic epidermolysis bullosa, allows testing of molecular therapies. In: Journal of Investigative Dermatology 127, Suppl. Suppl. 2, S78-S78 (2007).
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