Lubkemeier, I., R. P. Requardt, X. Lin, P. Sasse, R. Andrie, J. W. Schrickel, H. Chkourko, F. F. Bukauskas, J. S. Kim, M. Frank, D. Malan, J. Zhang, A. Wirth, R. Dobrowolski, P. J. Mohler, S. Offermanns, B. K. Fleischmann, M. Delmar and K. Willecke: Deletion of the last five C-terminal amino acid residues of connexin43 leads to lethal ventricular arrhythmias in mice without affecting coupling via gap junction channels. 108, 3, (2013).
url: http://www.ncbi.nlm.nih.gov/pubmed/23558439
Otten, J., P. F. M. van der Ven, P. Vakeel, S. Eulitz, G. Kirfel, O. Brandau, M. Boesl, J. W. Schrickel, M. Linhart, K. Hayess, F. J. Naya, H. Milting, R. Meyer and D. O. Furst: Complete loss of murine Xin results in a mild cardiac phenotype with altered distribution of intercalated discs. In: Cardiovascular Research 85, 4, 739-750 (2010).
http://edoc.mpg.de
The Max Planck Society does not take any responsibility for the content of this export.